2022/4/1

JAT Vol. 29 No.4が発刊されました

▶︎ Journal of Atherosclerosis and Thrombosis

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Reviews
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Atherosclerosis and Cardiovascular Diseases in Progeroid Syndromes
Hisaya Kato and Yoshiro Maezawa, J Atheroscler Thromb, 2022; 29: 439-447.

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Editorials
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Pitavastatin versus Atorvastatin: Potential Differences in their Effects on Serum Lipoprotein Lipase and Cardiovascular Disease
Junji Kobayashi, J Atheroscler Thromb, 2022; 29: 448-450.

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Original Articles
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Prevention of Cardiovascular Events with Pitavastatin is Associated with Increased Serum Lipoprotein Lipase Mass Level: Subgroup Analysis of the TOHO-LIP
Daiji Nagayama, et al.,J Atheroscler Thromb, 2022; 29: 451-463.

Three-Dimensional Fluid Dynamical Features of Coronary Plaque Rupture Provoking Acute Coronary Syndrome
Korehito Iida, et al.,J Atheroscler Thromb, 2022; 29: 464-473.

Association of Age with Mortality Rate after Femoropopliteal Endovascular Therapy for Intermittent Claudication
Mitsuyoshi Takahara, et al.,J Atheroscler Thromb, 2022; 29: 474-481.

Elevated Glycated Albumin in Serum Is Associated with Adverse Cardiac Outcomes in Patients with Acute Coronary Syndrome Who Underwent Revascularization Therapy
Jianwei Zhang, et al.,J Atheroscler Thromb, 2022; 29: 482-491.

Impact of H-Type Hypertension on Intraplaque Neovascularization Assessed by Contrast-Enhanced Ultrasound
Yuting Tan, et al.,J Atheroscler Thromb, 2022; 29: 492-501.

Prognostic and Practical Validation of ESC/EACTS High Ischemic Risk Definition on Long-Term Thrombotic and Bleeding Events in Contemporary PCI Patients
Hao-Yu Wang, et al.,J Atheroscler Thromb, 2022; 29: 502-526.

Leukocyte Count and Risks of Stroke and Coronary Heart Disease: The Circulatory Risk in Communities Study (CIRCS)
Jiaqi Li, et al.,J Atheroscler Thromb, 2022; 29: 527-535.

Clinical Importance of the LDL-C/Apolipoprotein B Ratio for Neointimal Formation after Everolimus-Eluting Stent Implantations
Naotaka Akutsu, et al.,J Atheroscler Thromb, 2022; 29: 536-550.

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Case Report
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A Resuscitated Case of Acute Myocardial Infarction with both Familial Hypercholesterolemia Phenotype Caused by Possibly Oligogenic Variants of the PCSK9 and ABCG5 Genes and Type I CD36 Deficiency
Ryo Nishikawa, et al.,J Atheroscler Thromb, 2022; 29: 551-557.

   
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